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Formation of IAA was considered very likely resulting from oxidase deamination of NMT (Barker et al., 1980), but was later on founded being also in part by immediate deamination by MAO (Barker et al., 1982). Pretreatment Using the MAO-I iproniazid in rat whole brain homogenates inhibited IAA development by eighty three%, NMT and DMT-NO development

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